Liraglutide reduces metabolic dysfunction, insulin resistance and lipotoxicity in the key metabolic organs in the pathogenesis of NASH.​​​​​​​
Hotair silence activates P16Ink4a and P14ARF signaling by enhancing miR-218 expression and suppressing Bmi-1 expression, resulting in the suppression of tumorigenesis in HCC​​​​​​​
EDIL3-mediated activation of TGF-β and ERK signaling could provide therapeutic implications for HCC.​​​​​​​
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